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The aggregation of amyloid-β peptide (Aβ) into amyloid fibrils and plaques is one culprit to Alzheimer’s disease pathogenesis. Once a small concentration of amyloid fibrils has formed through primary nucleation, a fibril-catalysed secondary nucleation takes over, leading to a close to exponential growth. Here, we have investigated the effect of five (J46, I48, J44, I68 and J7) out of twelve previo
